Jurkat, Clone E6-1
1
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Jurkat, Clone E6-1 originates from Jurkat-FHCRC, which is a derivative of the Jurkat cell line established from a 14-year-old male suffering from acute T-cell leukemia. This pseudodiploid human cell line maintains a prevailing chromosome count of 46 in 74% of cells, while 5.3% showcase polyploidy. Forged through incubation at 41°C and subsequent limiting dilution cloning over macrophages, Jurkat, 
Clone E6-1 displays escalated IL-2 production upon provocation by phorbol esters and T3 antigen-specific lectins or monoclonal antibodies.
Why choose Jurkat, Clone E6-1 from AcceGen?
Jurkat, Clone E6-1 boasts exceptional viability and quality, maintained through sterile conditions, free from bacterial, fungal, and mycoplasma contamination. Handled by skilled professionals, it undergoes precise incubation and cryopreservation. Rigorous quality control, including STR profile identification, ensures its reliability.
| Product Code | JurkatE6-1; Jurkat E6-1; Jurkat, Clone E6-1; Jurkat Clone E6-1; Jurkat (clone E6-1); JURKAT E-6.1; JURKAT E-61; Jurkat-CloneE61; Jurkat-E6; Jurkat E6; J.E6-1; E6-1 |
| Species | Human |
| Cat.No | ABC-TC0482 |
| Product Category | Tumor Cell Lines |
| Size/Quantity | 1 vial |
| Shipping Info | Dry Ice |
| Growth Conditions | 37 ℃, 5% CO2 |
| Source Organ | Blood |
| Biosafety Level | 1 |
| Storage | Liquid Nitrogen |
| Product Type | Human Lymphocytes Cell Lines |
When you publish your research, please cite our product as “AcceGen Biotech Cat.# XXX-0000”. In return, we’ll give you a $100 coupon. Simply click here and submit your paper’s PubMed ID (PMID).
FOR RESEARCH USE ONLY
Jurkat, Clone E6-1, stands as a versatile cornerstone in the realm of scientific inquiry, particularly within immune system disorder research, immunology, and immuno-oncology investigations. Its multi-faceted utility extends to facilitating IL-2 assays and playing a vital role in the intricate process of cloning studies. Beyond these fundamental applications, its significance amplifies as it unravels the complexities of cell-independent delivery mechanisms, scrutinizes the cytotoxic implications of the cytolethal distending toxin, and sheds light on the nuanced expressions of estrogen receptors, influenced by bisphenol A exposure, all expertly orchestrated within controlled in vitro settings.
