Human NINJ1 knockout cell line

Frequently Asked Questions

• What is the Human NINJ1 Knockout Cell Line?

  The Human NINJ1 Knockout Cell Line is a genetically engineered cell line where the NINJ1 gene has been disrupted or “knocked out” using CRISPR/Cas9 technology. This allows researchers to study the function of the NINJ1 gene by observing the effects of its absence on cellular processes.

• How is the Human NINJ1 Knockout Cell Line created?

  This cell line is created using CRISPR/Cas9 technology, a precise genome-editing tool that enables the targeted disruption of the NINJ1 gene. This results in the complete knockout of the gene, allowing for detailed studies of its role and function in various cellular processes.

• What are the research applications of the Human NINJ1 Knockout Cell Line?

  The Human NINJ1 Knockout Cell Line is used to investigate the biological functions and pathways involving the NINJ1 gene. It is valuable for studying neural regeneration, cell adhesion, inflammation, blood-brain barrier integrity, and apoptosis. These studies can contribute to the understanding of neurological and immunological diseases and the development of new therapeutic approaches.

• Why is the NINJ1 gene important?

  The NINJ1 gene plays crucial roles in several biological processes, including neural regeneration, cell adhesion, and immune response regulation. By studying the effects of NINJ1 knockout, researchers can gain insights into its function and involvement in disease mechanisms, potentially leading to new therapeutic targets for neurological and immune-related conditions.

• What are the advantages of CRISPR/Cas9-edited Human NINJ1 Knockout Cell Line?

  CRISPR/Cas9-edited Human NINJ1 Knockout Cell Line offers several advantages, including high specificity and efficiency in gene editing, allowing for precise disruption of the NINJ1 gene. This facilitates the study of gene function and interaction in a controlled environment, providing reliable data for understanding NINJ1’s role in cellular processes and disease states.